In this blog you will find the correct answer of the Coursera quiz Diabetes a Global Challenge Coursera Answers mixsaver always try to bring the best blogs and best coupon codes
Week- 1
Epidemiology of Diabetes
1.
Question 1
How is the trend in occurrence of diabetes worldwide?
1 point
- Increasing
- Decreasing
- Stable
2.
Question 2
How is diabetes defined?
1 point
- A disease in which insulin works too effectively
- A disease in which glucose is excreted with the urine
- A disease in which the pancreas secretes no or too little insulin
3.
Question 3
What is the difference between type 1 and type 2 diabetes?
1 point
- Type 1 diabetes occurs only among children and type 2 only among adults
- In Type 1 diabetes, the pancreas produces no insulin, whereas in type 2 diabetes, the insulin produced does not work adequately
- Type 2 diabetes follows after some years with type 1 diabetes
4.
Question 4
How many different types of diabetes are there?
1 point
- Three main types and multiple more rare subtypes
- Two, type 1 and type 2 diabetes
- Three, type 1, type 2 and type 3 diabetes
5.
Question 5
Which region of the world is most severely affected by diabetes?
1 point
- Europe and Russia
- The Middle East and the Far East
- North, Central and South America
6.
Question 6
How is the age distribution at onset of type 2 diabetes?
1 point
- Type 2 diabetes patients get the disease shortly before they die from other diseases
- Type 2 diabetes is mainly emerging in late adulthood
- Type 2 diabetes emerges mainly during childhood and young adulthood
7.
Question 7
How can the global epidemic of diabetes lead to a public health crisis?
1 point
- Not enough medicine is produced to supply all the diabetic patients with what they need
- Nothing can be done about it
- The diabetic patients demand a medical care that many countries cannot afford to provide
8.
Question 8
What is impaired glucose tolerance?
1 point
- Glucose in the blood has been chemically modified and therefore cannot be tolerated
- Intake of glucose cannot be tolerated and leads to sickness
- Even though the pancreas secretes more insulin, it cannot keep the glucose levels down to normal levels
9.
Question 9
How is overweight affecting risk of diabetes?
1 point
- Increases the risk
- Decreases the risk
- Does not affect the risk
Week- 2
Obesity and Prevention of Diabetes
1.
Question 1
How much physical activity do major health agencies recommend for adults?
1 point
At least 75 minutes of moderate-intensity or at least 35 minutes of vigorous-intensity aerobic physical activity per week – or a combination. Additionally, muscle-strengthening activities should be performed on 2 or more days a week.
At least 300 minutes of moderate-intensity or at least 150 minutes of vigorous-intensity aerobic physical activity per week – or a combination. Additionally, muscle-strengthening activities should be performed on 4 or more days a week.
At least 150 minutes of moderate-intensity or at least 75 minutes of vigorous-intensity aerobic physical activity per month – or a combination. Additionally, muscle-strengthening activities should be performed on 2 or more days a month.
At least 150 minutes of moderate-intensity or at least 75 minutes of vigorous-intensity aerobic physical activity per week – or a combination. Additionally, muscle-strengthening activities should be performed on 2 or more days a week.
2.
Question 2
Compared to adults, major health agencies recommend children to be…
1 point
… more physically active
… less physically active
… as physically active
3.
Question 3
The ‘state-of-the-art’ test for assessment of physical fitness is by…
1 point
… heart rate monitoring
… an activity questionnaire
… quantification of maximal oxygen uptake by indirect calorimetry during a biking or running test to exhaustion
… the doubly-labelled water method
4.
Question 4
Health benefits of being physically active include…
1 point
… a decrease in insulin sensitivity and a higher heart stroke volume
… decreases in plasma HDL-cholesterol and triglyceride
… an increase in insulin sensitivity and a decrease in blood pressure
… stronger tendons and softer skin
5.
Question 5
The influence of physical training on energy balance is more complicated than previously thought as…
1 point
… compensatory mechanisms might come into play
… seasonal changes affect both physical training and energy balance
… smoking is increased by physical training
… caffeine intake is reduced by physical training
6.
Question 6
Approximately 50 kcal are expended by…
1 point
… playing with the children for two hours
… a 60-minute workout in the fitness center
… sleeping on the couch for 8 hours
… a 10-minute walk during the lunch break
7.
Question 7
Metabolic health benefits of endurance training include…
1 point
… an increase in HOMA-IR
… an increase in waist circumference
… an increase in peripheral insulin sensitivity
… a decrease in HDL-cholesterol
8.
Question 8
People with diabetes and those at risk for diabetes…
1 point
… cannot improve glycemic control by physical exercise
… should abstain from physical activity
… show decreases in peripheral insulin sensitivity after endurance training
… should be physically active on a regular basis
Week- 3
Physiological Regulation of Plasma Glucose
1.
Question 1
What is the range of normal plasma concentrations?
1 point
3-12 mmol/l
5-7 mmol/l
4-10 mmol/l
2.
Question 2
Which is the correct statement? The glucose pool is:
1 point
Extracellular volume multiplied the by plasma glucose concentration
Intracellular volume multiplied by the plasma glucose concentration
Total body water multiplied by the plasma glucose concentration
3.
Question 3
Which statement is false?
1 point
glycerol may be converted into glucose
Amino acids may be converted into glucose
Lipids may be converted into glucose
4.
Question 4
Which is the correct statement? How can beta cells sense extracellular glucose concentrations?
1 point
Because they express the glucose transporter GLUT-4
Because intracellular glucose is converted to ketone bodies
Because of the high Km of glucokinase
5.
Question 5
Which statement is false?: Insulin secretion is stimulated by…
1 point
Arginine
Sympathetic nerves
Parasympathetic nerves
6.
Question 6
Which statement is false?
1 point
The incretin effect is responsible for up to 70 % of the postprandial insulin secretion
The incretin effect is responsible for keeping up insulin secretion in type 2 diabetes
The incretin effect keeps post prandial glucose excursions low regardless of the carbohydrate load
7.
Question 7
Which statement is correct? In the liver, insulin…
1 point
stimulates glycogenolysis
Stimulates glycolysis
Stimulates gluconeogenesis
8.
Question 8
Which statement is correct: Glucagon stimulates…
1 point
glycogenesis
glycolysis
gluconeogenesis
9.
Question 9
Which is the correct statement? The limiting factor for prevention of deadly hypoglycaemia during prolonged fasting is:
1 point
Cortisol-stimulated provision of gluconeogenic amino acids
Glucose formation from ketone bodies
Glucagon-stimulation of gluconeogenesis
10.
Question 10
Which is the false statement? During exercise plasma glucose does not fall very much because:
1 point
Glycogenolysis in the muscles keeps it up
Insulin secretion is inhibited
Gluconeogenesis from lactate is increased
Week- 4
The Incretins
1.
Question 1
What is the name of the two incretin hormones?
1 point
GLP-1 and GIP
GLP-1 and oxyntomodulin
GLP-1 and PYY
Glucagon and GLP-1
2.
Question 2
Where is the incretin hormones secreted from?
1 point
The brain
The skin
The gut
The hair
3.
Question 3
What is the name of the cell type secreting the incretin hormones?
1 point
Goblet cell
Enterocyte
Enteroendocrine
4.
Question 4
GLP-1 is derived from the prohormone called
1 point
Glucagon
Progip
Proglp-1
Proglucagon
5.
Question 5
Proconvertase 1/3 is responsible for the differential splicing of proglucagon to GLP-1 in the pancreas and not in the gut?
1 point
Yes
No
6.
Question 6
Where is GLP-1 primarily degraded?
1 point
In the brain
The capillaries draining the gut
The capillaries of the gut and the liver
The blood
7.
Question 7
GLP-1 affects
1 point
Glucose regulation
Appetite regulation
Gastric emptying
All of the options
8.
Question 8
GLP-1 responses are often impaired in T2D subjects?
1 point
Yes
No
9.
Question 9
Why are GLP-1 receptor agonists used as therapy in type 2 diabetes?
1 point
Because GLP-1 inhibits glucagon secretion
Because GLP-1 inhibits appetite
All of the options
Because GLP-1 stimulate glucose induced insulin secretion
10.
Question 10
Inhibition of the DPP-4 enzyme can be used as therapy of type 2 diabetes?
1 point
Yes
No
11.
Question 11
GLP-1 receptor agonists are administered
1 point
Intravenously
Orally
Subcutaneously
Rectally
12.
Question 12
How many per cent of subjects can skip their diabetes medication for their type 2 diabetes after gastric bypass surgery?
1 point
60
20
100
40
10
80
30
13.
Question 13
Which factors may be important for the remission of type 2 diabetes after gastric bypass
1 point
Malabsorption of lipids
Cytokines
Malabsorption of carbohydrates
Gut hormones
14.
Question 14
Is the release of gut hormones (e.g. GLP-1, PYY and Oxyntomodulin) increased after gastric bypass?
1 point
No
It has not been investigated
Yes
15.
Question 15
What happens to insulin secretion if gastric bypass subjects receive a GLP-1Receptor antagonist during a meal challenge?
1 point
The insulin secretion is increased
The insulin secretion does not change
The insulin secretion is diminished
Week- 5
Clinical manifestation of Diabetes and treatment
1.
Question 1
Choose the correct answer:
1 point
The diabetes epidemics occur as a result of a true increased incidence of the disease in all countries.
Lowering of diagnostic criteria for T2D has contributed to the increased incidence of the disease.
The largest increase in future T2D incidence is expected to take place in affluent Western societies.
2.
Question 2
Choose the correct answer:
1 point
The 2 hours cut-off level for the T2D diagnosis based on an oral glucose tolerance test was based on association studies showing increased risk of microvascular complications with levels above the diagnostic threshold.
Patients with T2D do not have increased risk of cardiovascular disease after correction for the ambient plasma glucose levels.
Lowering of the cut-off criteria for fasting plasma glucose in T2D diagnosis is based on randomized trials providing conclusive evidence that intensive glucose control lower cardiovascular mortality.
3.
Question 3
Choose the correct answer:
1 point
The idea of T2D being a ”category error” reflect inappropriate knowledge about the origin and underlying pathophysiological mechanisms in T2D.
The idea of T2D as a ”category error” reflect that it resemble T1D more than generally recognized.
The idea of T2D being a ”category error” reflects inappropriate attention to the known increased risk of cardiovascular complications associated with the disease.
4.
Question 4
Choose the correct answer:
1 point
The association between T2D, hypertension and dyslipidaemia may be explained by common factors occurring in foetal life influencing the risk of all of these phenotypes
The association between T2D, hypertension and dyslipidaemia is based on a common genetic origin illustrated by clustering of these states of disease in families.
The association between T2D and cancer is due to glucose lowering agents including insulin causing cancer.
5.
Question 5
Choose the correct answer:
1 point
All patients with T2D benefit to the same extent from intensive versus conventional glucose lowering treatment.
Individualized treatment of patients with T2D means that those who are expected to benefit most from the different treatment modalities will get increased access to this from health care providers.
Individualized treatment of patients with T2D will result in increased health care expenses.
6.
Question 6
Choose the correct answer:
1 point
Intensive glucose lowering treatment does not reduce cardiovascular mortality in T2D.
Multifactorial intervention does not reduce mortality from cardiovascular disease in T2D.
The legacy effect means that intensive glucose lowering treatment initiated immediately after the T2D diagnosis will ensure that the patient does not develop micro- or macro vascular complications.
7.
Question 7
Choose the correct answer:
1 point
All patients with T2D exhibit absolute defects of multiple organs including the liver, muscle, adipose tissue, the pancreatic beta cell, as well as gut incretin hormone functions.
Impaired left ventricular heart function increase the risk of developing T2D.
Genetic factors do not influence the risk of developing T2D.
8.
Question 8
Choose the correct answer:
1 point
A DXA scanning provides accurate information about the amount of intra-abdominal (visceral) fat.
Lack of expandability of the subcutaneous adipose tissue may impact adversely on glucose homeostasis due to fat accumulation and lipotoxicity in muscle, fat and the pancreatic beta cell.
Neuro-cognitive dysfunctions in T2D including depression occur solely as a result of the elevated plasma glucose level.
9.
Question 9
Choose the correct answer:
1 point
Hales and Barker were the first scientists who proposed that adverse early life conditions increases the risk of developing adult non-communicable cardiometabolic diseases.
Low birth weight is to a large extent explained by known T2D susceptibility genes causing low pancreatic insulin secretion and subsequently reduced growth in foetal life.
The ”thrifty phenotype hypothesis” provides a potential explanation for the multiple organ dysfunctions in T2D.
10.
Question 10
Choose the correct answer:
1 point
Lifestyle interventions improving diet and physical activity habits provide sustainable prevention of T2D as well as its micro- and macro vascular complications.
None of the currently used glucose lowering agents reduces the risk of diabetic vascular complications beyond their glucose lowering effects
Metformin and GLP-1 agonists are the only glucose lowering drugs which conclusively have been shown to reduce cardiovascular mortality in T2D.
Week- 6
Inflammatory Beta-cell Destruction in Diabetes
1.
Question 1
What is the cause of diabetes?
•
•
•
1 point
Most diabetes is due to mutations in the insulin signalling pathway causing insulin resistance
Most diabetes is caused by reduced tubular glucose reabsorption causing glucosuria
Most diabetes is due to relative or absolute insulin deficiency
2.
Question 2
What is the reason that insulin-resistant people progress from normal glucose tolerance to impaired glucose tolerance and diabetes?
1 point
Because of increased glucagon secretion from the pancreatic islets
Because of increased sucrose absorption from the gut
Because insulin secretion fails to compensate for insulin resistance
3.
Question 3
Insulin secretion in diabetes is…
1 point
reduced only because of impaired beta-cell function
increased because of increased beta-cell mass
reduced because of impaired beta-cell function and mass
4.
Question 4
What are the immune-cells involved in beta-cell killing in T1 diabetes?
1 point
B-lymphocytes and Th2 cells
Macrophages and cytotoxic T-cells
Dendritic cells and Th2 cells
5.
Question 5
How are macrophages believed to kill beta-cells in diabetes?
1 point
By direct cell-to-cell contact (‘a deadly kiss’)
By recognition of danger-associated molecular beta-cell patterns causing membrane perforation
By secretion of inflammatory mediators
6.
Question 6
How are T-cells believed to kill beta-cells in T1 diabetes?
1 point
By the release of bradykinins, histamine and prostaglandins
By the secretion of nitric oxide radicals
By the Fas/FasL or perforin/granzyme pathway
7.
Question 7
How do metabolic factors affect insulin sensitive tissues and beta-cells in T2 diabetes?
1 point
By causing low-grade inflammation and gluco-lipotoxicity in the cells
By causing vascular dysfunction, reduced blood flow and ischemia in the tissues
By causing accumulation of glycogen and protein deposits in the cells
8.
Question 8
What is a common consequence of islet inflammation in T1 and T2 diabetes?
1 point
Beta-cell necrosis
Alpha cell apoptosis
Beta-cell apoptosis
9.
Question 9
How do beta-cell sense inflammatory signals?
1 point
By beta-adrenergic receptors
By cytokine and Toll-like receptors
By mitochondrial oxidation of the increased glucose levels
10.
Question 10
What are the perspectives for future antiinflammatory therapies in T1 diabetes?
1 point
To combine T-cell inhibitors and activators of the innate immune system
To combine T-cell activators and inhibitors of the innate immune system
To combine T-cell inhibitors and inhibitors of the innate immune system
11.
Question 11
What are the perspectives for future antiinflammatory therapies in T2 diabetes?
1 point
Antiinflammatory therapy given from birth in children at genetic risk for T2 diabetes
Short courses of antiinflammatory therapy may break glucose-driven islet inflammation
Long-term cytokine biologics as monotherapy from onset of T2 diabetes
Week- 7
Genetic Aspects of Diabetes
1.
Question 1
Type 2 diabetes is a complex disease with an important genetic component. The prevalence of type 2 diabetes in the adult European population is about 5-10%. What is the lifetime risk for the offspring for developing type 2 diabetes:
1 point
90-100%
5-10%
30-40%
2.
Question 2
Heritability measures the fraction of phenotype variability that can be attributed to genetic variation. What is the heritability of body mass index (BMI):
1 point
5-10%
10-20%
>50%
3.
Question 3
The Human Genome Project has revealed the approximate number of protein coding genes in the human genome. That is:
1 point
>100,000 genes
About 5,000-10,000 genes
About 20,000-25,000 genes
4.
Question 4
A 2 month old child is diagnosed with diabetes. The child has likely:
1 point
Neonatal diabetes
Type 1 diabetes
Type 2 diabetes
5.
Question 5
Do we need to perform a genetic test in a 2 month old child diagnosed with diabetes:
1 point
No, the diagnosis is most likely type 2 diabetes which could be treated with metformin
Yes, a genetic test will often reveal the correct diagnosis and will allow for counseling and optimal treatment choices
No, the diagnosis is most likely type 1 diabetes which should be treated with insulin
6.
Question 6
Maturity Onset Diabetes of the Young (MODY) is a common form of monogenic diabetes accounting for up to:
1 point
5-10% of all cases of diabetes
1-2% of all cases of diabetes
>10% of all cases of diabetes
7.
Question 7
Children diagnosed with GCK-diabetes (MODY2) should be treated with:
1 point
No treatment
Metformin
Sulphonylurea
8.
Question 8
Patients diagnosed with HNF1A-diabetes (MODY3) with elevated glucose levels should be offered treatment with:
1 point
Sulphonylurea
Metformin
Insulin
9.
Question 9
Genome wide association studies have revealed a number of common variants which increases the risk of type 2 diabetes. How many type 2 diabetes risk variants have been reported:
1 point
About 10
About 20
>50
10.
Question 10
Is it important to perform genetic testing for monogenic forms of diabetes in selected patient groups:
1 point
Yes, testing makes the right diagnosis, helps counseling and can guide choice of treatment
No, following present treatment guidelines are sufficient
No, circulating biomarkers can be used to diagnose specific forms of diabetes which makes the right diagnosis, helps counseling and can guide choice of treatment
Week- 8
Translational Aspects
1.
Question 1
Translational
science is
1 point
from
diabetes to treatment
from
bedside to lab bench
from lab bench to bedside
2.
Question 2
During an OGTT in subjects with mutations on their KCNQ1 receptor the consequence was
1 point
Hyperglycemia
Hypoglycemia
Euglycemia
3.
Question 3
Patients with
mutation in their GIP receptor had
1 point
Lower fracture risk
higher fracture risk
no difference
Week- 9
Development of antidiabetic agents
1.
Question 1
The response of GI hormones during a meal is
1 point
The secretion of GI hormones e.g. GLP-1 is reduced
No response
The secretion of GI hormones e.g. GLP-1 is increased
2.
Question 2
Ghrelin levels increase during intake of a meal
1 point
True
False
Nothing happens to ghrelin
3.
Question 3
The Gut microbiota may assist the conversion of carbohydrates (fibres) into
short chain fatty acids (SCFA)
1 point
False
True
4.
Question 4
Gut hormones may exert
1 point
Paracrine functions
All of the mentioned options
Neuronal activation (e. g. sensory vagal afferents)
Endocrine functions
5.
Question 5
What does residual capacity of the enteroendocrine cells means?
1 point
That they have residual ATP
That the enteroendocrine cells may still be able to release hormone upon additional stimulation by e.g. nutrients (a bigger meal )
That more GLP-1 is being secreted
Nothing, just an expression
6.
Question 6
Which of the following hormones may influence satiety in humans?
1 point
PYY
Ghrelin
CCK
GLP-1
All of the mentioned options
7.
Question 7
The melanocortin receptor 4 is not associated with obesity?
1 point
False
We don’t know
True
8.
Question 8
What is the meaning of the expression: the gut-brain axis?
1 point
That activities in the gut may influence brain activities
That it is the gut which controls our emotions?
Nobody knows
That the brain controls the gut
9.
Question 9
Which receptor(s) does Amferpramone block?
1 point
DAT
All of the mentioned
NET
SERT
10.
Question 10
What is the main effect of Orlistat?
1 point
Inhibition of the pancreatic lipase
Inhibition of NET
Inhibition of adrenergic receptors
11.
Question 11
Can GLP-1 analogs been used in treatment of obesity?
1 point
Yes, but the GLP-1 analogue need to be resistant to inactivation by DPP-4
No
Yes
12.
Question 12
Can mixture of more than one peptide be beneficial in treatment of obesity?
1 point
No
Yes, use of more than one peptide can be beneficial in treatment of obesity
13.
Question 13
Glucagon together with GLP-1 could be an important co-agonist in treatment of diabetes and obesity?
1 point
True
False
Glucagon does not regulate satiety
Maybe
Week- 10
Stem Cell Based Therapy of Diabetes
1.
Question 1
Diabetes late complications distinguish type 1 diabetes from type 2 diabetes.
1 point
True
No, it is the reverse
No, both forms of diabetes can result in similar severe late complications.
2.
Question 2
Obese insulin-resistant people with normal blood-glucose levels have…
1 point
… a reduced beta cell mass compared to weight-matched T2D patients
… an unchanged beta cell mass compared to weight-matched T2D patients
… a greater beta cell mass compared to weight-matched T2D patients
3.
Question 3
Why is a constant blood-glucose important? (multiple answers are allowed in this question)
1 point
It secures constant energy to the brain
It prevents many of the diabetes late complications to develop
It allows you to effectively secrete glucose into the urine
4.
Question 4
Which symptom is considered amongst the worst when suffering from insulin-dependent diabetes?
1 point
Normoglycemia
Hypoglycemia
Hyperglycemia
5.
Question 5
The “Edmonton protocol” (published in yr 2000) refers to (multiple answers are allowed in this question):
1 point
Organ donor islet transplantation of T1D that effectively lead to strong reduction of recurrent hypoglycemic episodes
Organ donor islet transplantation of T1D patients to reach a situation where need for insulin injections may be strongly reduced or not needed
Whole pancreas transplantation to cure diabetes.
6.
Question 6
Circulating C-peptide levels in T1D patients indicate (multiple answers are allowed in this question):
1 point
That they will have a greater probability to develop severe complications
That patients still produce some endogenous insulin
Presence of active, living beta cells that have not yet been destroyed by autoimmunity
7.
Question 7
What is the major mechanism used by our body to secure maintenance of adult stem cells (or tissue stem cells) throughout life?
1 point
Asymmetrical cell division
Symmetrical cell division
Neo-formation
8.
Question 8
From which germ layer are the insulin producing pancreatic beta cells are derived?
1 point
Endoderm
Ectoderm
Mesoderm
9.
Question 9
Encapsulation of therapeutic beta cells serve the following purpose(s) (multiple answers are allowed in this question):
1 point
To protect against attack from the patient’s immune system
Eliminates the need for anti-rejection therapy
To shield the cells against oxidative stress
10.
Question 10
The pancreas is 98% exocrine and 2 % endocrine – what does it mean?
1 point
The endo- and exocrine cells secrete their products into the blood stream
The endocrine products are secreted into the blood stream while the exocrine products are secreted via the duct to the intestine
The endo- and exocrine cells secrete their products via the ductal system to the intestine
Week- 11
Exercise – how does it work?
1.
Question 1
What kind of fat distribution is most dangerous in relation to the development of Type 2 Diabetes?
1 point
2.
Question 2
Melissa has been severely injured in an accident and spends 3 weeks in bed to recover. What is the impact on her physical work capacity if you measure in years of ageing?
1 point
Equivalent to 10 years of ageing
Equivalent to 20 years of ageing
Equivalent to 30 years of ageing
Equivalent to 40 years of ageing
3.
Question 3
Ann, Beatrice and Cathrine are sisters. Who is most at risk of developing type 2 diabetes?
1 point
Ann
Age: 51
Weight: 74
BMI: 29
Physical activity: takes bus and train to work, walks a lot at work, plays tennis once a week.
Beatrice
Age: 54
Weight: 79
BMI: 27
Physical activity: drives to work, no regular exercise
Catherine
Age: 55
Weight: 80
BMI: 31
Physical activity: bikes to work, does yoga twice a week.
4.
Question 4
What are the effects of the myokines that are secreted from skeletal muscles during exercise?
1 point
They provoke an inflammatory state in some organs.
They have no effect on organs, they help build muscle tissue.
They have beneficial effects on several organs.
5.
Question 5
Which of the following proteins are secreted from an exercising leg?
1 point
IL-4
Glucagon
Adiponectin
Follistatin
GLP-1
IL-7
IL-6
6.
Question 6
How may exercise prevent tumour growth?
1 point
Exercise increases the number of immune cells and directs them towards the tumour.
Exercise does not prevent tumour growth
The myokines secreted in respons to exercise directly attacks cancer cells.
7.
Question 7
What are the effects of inflammation?
1 point
Inflammation enables us to fight infections.
Inflammation increases levels of pro-inflammatory cytokines.
Inflammation increases levels of myokines.
There are no effects of inflammation.
8.
Question 8
Does the location of fat affect systemic inflammation?
1 point
Yes, because the location of fat affects the secretion of myokines.
No, fat affects inflammation independent of location.
Yes, because (in particular) visceral fat induces low-grade inflammation.
9.
Question 9
Why is exercise beneficial for a wide range of chronic illnesses like Dementia, Cancer, Depression and Type 2 Diabetes?
1 point
Exercise reduces systemic inflammation and provides proper navigation for immune cells to find cancerous cells.
Exercise is not beneficial for chronic illnesses.
Exercise only have acute effects on our health and not long-term effects.
I understand that submitting another’s work as my own can result in zero credit for this as
Week- 11
Peer-graded Assignment: Peer graded assignment
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